Muscular Dystrophy

Scientists have developed a new procedure to increase the length of human telomeres. This increases the number of times cells are able to divide, which may point the way to treating various age-related disorders – or even muscular dystrophy.

A new study suggests that a novel combination of the drugs currently used to control high blood pressure in the one in 3,500 young males suffering from Duchenne muscular dystrophy could slow the decline in heart function earlier on, and in promising new ways.

A new technique for gene editing that removes a mutation leading to a mouse model of Duchenne muscular dystrophy could have far-reaching consequences in the treatment of muscular dystrophy in people.

Researchers have been able to double muscle strength and endurance in mice and worms through a single genetic tweak.

The human protein biglycan significantly slows muscle damage and improves function in mice with Duchenne Muscular Dystrophy by causing utrophin, a natural muscle-building protein prevalent in young children, to collect in muscle cell membranes.

REX is a robotic exoskeleton that puts wheelchair users back on their feet, enabling a person to stand, walk and go up and down stairs and slopes.